Download PDF by M. Brizzio : Acute Coronary Syndromes

By M. Brizzio

ISBN-10: 9533078278

ISBN-13: 9789533078274

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Thrombus is composed of platelet thrombus (P) and mixed thrombus + fibrin-rich thrombus (P+M). hematoxylin and eosin. 30 Acute Coronary Syndromes Fig. 4. Serial section of figure 1. Platelet thrombus is deeply stained than mixed thrombus + fibrin-rich thrombus, and adhered on the inner side of mixed thrombus + fibrin-rich thrombus. Immunochemistry for CD42b. Fig. 5. Serial section of figure 1. Fibrin mesh is contained in mixed thrombus + fibrin-rich thrombus but not in platelet thrombus . phosphotungstic acid hematoxylin stain.

16. Scavenger receptor A positive cells in peripheral blood (SRA index: 187/10HPFs). Table 2. The scattered plots of the SRA index of controls, AMI, UA. Physiopathology of the Acute Coronary Syndromes 39 9. Differentiation of SRA+ cells in PB It is reported that freshly isolated blood monocytes were negative for SRA 25; however, a few reports have indicated that SRA-monocytes can differentiate into SRA+ cells in PB by various stimuli 32, 33, 34. SRA+ cells were observed in all control cases. We , therefore, - surmised that a small number of SRA monocytes differentiated into SRA+ cells in PB of healthy men and women by various stimuli.

5). We considered two possibilities on the pathophysiologic mechanisms underlying the differentiation of SRA+ cells in patients with STEMI (myocardial infarction or plaque disruption, fissure or erosion). With the recognition that atherosclerosis is an inflammatory process 36, h-CRP has been evaluated as a potential tool for predicting the risk of AMI 37, 38, 39, and CK and CK-MB are good markers of myocardial injury. Macrophages are known to arise from monocytes, and become larger over twice as large as monocytes with differentiation.

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Acute Coronary Syndromes by M. Brizzio


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